Cardiovasc Res (2011) 90 (2): 276-284. doi: 10.1093/cvr/cvq376
Ana Planavila1,2,*, Roser Iglesias1,2, Marta Giralt1,2 and Francesc Villarroya1,2
1Departament de Bioquímica i Biologia Molecular, Institut de Biomedicina de la Universitat de Barcelona (IBUB), Universitat de Barcelona, Avda Diagonal 645, Barcelona E-08028, Spain
2CIBER Fisiopatología de la Obesidad y Nutrición (CIBEROBN), Barcelona, Spain
"Collectively, these findings reveal a major involvement of the Sirt1–PPARα interaction in the protective role of Sirt1 against cardiac hypertrophy."
Friedreich Ataxia and close related scientific news. Topics related to rare diseases.
Monday, April 18, 2011
Friedreich's ataxia: Past, present and future
doi:10.1016/j.brainresrev.2011.04.001
Daniele Marmolino,
Laboratoire de Neurologie experimentale, Universite Libre de Bruxeles (ULB), Route de Lennik 808, Campus Erasme - 1070 Bruxelles, Belgium
Keywords: Friedreich's ataxia; Frataxin; GAA; Iron; Fe-S clusters; Oxidative stress; FRDA pathogenesis; frataxin function; new treatments; animal and cellular models.
Daniele Marmolino,
Laboratoire de Neurologie experimentale, Universite Libre de Bruxeles (ULB), Route de Lennik 808, Campus Erasme - 1070 Bruxelles, Belgium
Keywords: Friedreich's ataxia; Frataxin; GAA; Iron; Fe-S clusters; Oxidative stress; FRDA pathogenesis; frataxin function; new treatments; animal and cellular models.