Shortening trinucleotide repeats using highly specific endonucleases: a possible approach to gene therapy?

Shortening trinucleotide repeats using highly specific endonucleases: a possible approach to gene therapy?. Guy-Franck Richard; Trends in Genetics, Available online 2 March 2015, ISSN 0168-9525, http://dx.doi.org/10.1016/j.tig.2015.02.003.

Mechanism of action of 2-aminobenzamide HDAC inhibitors in reversing gene silencing in Friedreich’s ataxia

Mechanism of action of 2-aminobenzamide HDAC inhibitors in reversing gene silencing in Friedreich’s ataxia. Soragni E, Chou CJ, Rusche JR and Gottesfeld JM (2015). Front. Neurol. 6:44. doi: 10.3389/fneur.2015.00044

Friedreich's Ataxia (FRDA) is an extremely rare cause of Autosomal Recessive ataxia in Chinese Han population

Friedreich's Ataxia (FRDA) is an extremely rare cause of Autosomal Recessive ataxia in Chinese Han population.
Junsheng Zeng, Junling Wang, Sheng Zeng, Miao He, Xianfeng Zeng, Yao Zhou, Zhen Liu, Hong Jiang, Beisha Tang; Journal of the Neurological Sciences, Available online 6 March 2015, doi:10.1016/j.jns.2015.03.002