Features of long-axis dysfunction in FRDA include: (i) a smaller contraction amplitude, which is partly attributable to a smaller LVEDL, and therefore has both structural and functional elements, (ii) smaller early diastolic excursion which is fully accounted for by smaller long-axis contraction, and (iii) lack of any compensatory increase in LA contraction, which is consistent with the presence of a concomitant LA myopathy.