Mitochondrial dysfunction induced by frataxin deficiency is associated with cellular senescence and abnormal calcium metabolism

Mitochondrial dysfunction induced by frataxin deficiency is associated with cellular senescence and abnormal calcium metabolism. Bolinches-Amorós A, Mollá B, Pla-Martín D, Palau F and Gonzalez-Cabo P; Front. Cell. Neurosci. 8:124. doi: 10.3389/fncel.2014.00124

Keywords: Friedreich Ataxia, frataxin, Mitochondrial dysfunction, cellular senescence, Autophagy, calcium metabolism, ER-stress

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